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High heterogeneity was observed in both subgroups (Cognitive Impairment: I 2 = 86%

(54) found that administering GIPR antagonist to healthy humans reduced postprandial insulin secretion and increased glucose excursions more than administering GLP-1R antagonist (GLP-1RA), indicating that endogenous GIP may play a more significant role in stimulating insulin secretion and reducing postprandial plasma glucose excursions compared to endogenous GLP-1

Its protective effects against oxidative stress can increase the overall antioxidant capacity of the cells []

Stomach regulates energy balance via acylated ghrelin and desacyl ghrelin

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