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FIGURE 5 Inhibition of AMPK Constrains the Cardio-Protective Effects of LCA Supplementation in vitro To investigate whether AMPK is crucial in LCA-conferred cardio-protection, we used PA, a saturated fatty acid, to mimic obesity in vitro, and CC, a pharmacological inhibitor of AMPK, to block AMPK signaling in primary atrial cardiomyocytes (Figure 6A)

2012 7 doi: 10.1371/journal.pone.0033414

Fc fusion to glucagon-like peptide-1 inhibits degradation by human DPP-IV, increasing its half-life in serum and inducing a potent activity for human GLP-1 receptor activation

It is important to note that this dosing protocol is provided as an example and should be adapted to the specific research question, study design, and regulatory requirements of the intended investigation