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It is proposed that the non-ubiquitinated NRF2 would remain bound to the KEAP1-Cul3 complex and therefore prevent the newly translated NRF2 from inhibition by the E3-ubiquitin complex, thereby promoting NRF2 accumulation in the nucleus [17]

Now, there are more potent anti-inflammatory drugs and biological agents, which may limit the application of AMDs in RA treatments ( 5.3 Modulators of STING Recent studies have delved into compounds that modulate STING activity and their potential utilization in the treatment of RA (70)

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Protocols at Perfect B in Doral are individualized, not templated

the MannWhitney U test for independent samples and the Wilcoxon signed rank test for paired samples