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However, some children develop significant obesity, insulin resistance, PMOS (formerly PCOS), prediabetes, or other metabolic conditions where lifestyle changes alone may not be enough

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Ethionamide requires activation by the mycobacterial enzyme flavin monooxygenase (EthA) and the transcriptional repressor EthR to form ethionamide sulfoxide, which binds to NAD + and inhibits the enoyl-acyl carrier protein reductase (InhA), thereby blocking mycolic acid synthesis, responsible for cell death (Dover et al., 2007)