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Abstract The global incidence of steatotic liver disease (SLD), driven by metabolic dysfunction-associated steatotic liver disease (MASLD), alcohol-associated liver disease (ALD), and the synergistic metabolic dysfunction and alcohol-associated liver disease (MetALD), is fueling a rapid rise in hepatocellular carcinoma (HCC)

When 0.25 g/mL biotinylated FcRn were used at pH 7.4, the receptor was pre-incubated with alkaline phosphatase (AP) conjugated streptavidin (1:5000 in PBS/T/S) to increase sensitivity