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StatPearls [Internet] 94

Utilizing HPLCMS/MS, metabolic intermediates in the malateaspartate shuttle, urea cycle, glucosealanine cycle and beta-alanine, aspirate, and tryptophan breakdown pathways were found to differ significantly between controls and patients with ASD

When the glutathione levels in regulatory T cells went down, immune activation increased, and the mouse model began to reject tumors

Figure 4 depicts the pathogenesis of PsO highlighting key pathways such as immune cell activation, cytokine signaling (IL-17, IL-23, TNF-), and keratinocyte proliferation

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