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While metabolic reprogramming may serve as an adaptive response to pathology, it also opens new targets for ferroptosis-directed therapy

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Bisphenol-A treatment also reduces the number of differentiating neurons from an immortalized human NSPC line known as ReNcell VM ( Recent investigations have demonstrated that bisphenol-A affects mitochondrial biology in hippocampal NSPCs, thus leading to increased oxidative stress and apoptosis (Figure 3)

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