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The incretin effect of GLP-1 is largely due to its direct actions on pancreatic beta and delta cells, but another vagal signaling pathway may exist that enhances incretin effects [33]

The calcium-dependent binding of CAO to ANT induces a conformational change (Halestrap et al., 2002) which results in the opening of the mitochondrial permeability transition pore (MPTP) and allows the equilibration of small solutes and the release of pro-apoptotic proteins from the intermembrane space

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However, regular or high-dose NSAID use demands periodic kidney function monitoring via serum creatinine and glomerular filtration rate (eGFR) testing