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Since its administration generally leads to a decrease in serum calcium, a close follow-up is required, at least initially, to avoid severe hypocalcemia with possible adverse clinical consequences

Continuous IGF-1 LR3 administration beyond 810 weeks triggers compensatory IGF-1 receptor downregulation cells reduce surface receptor density in response to chronic overstimulation, diminishing response magnitude

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No lethal dose has been established in animal studies, and the peptide appears to have wide safety margins