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Although the mechanism by which propofol can induce apoptosis in lymphocytes is not yet understood, some studies indicate that this process is transiently activated in the early postoperative period and that one factor responsible for this is the generation of ROS, leading to accelerated loss of lymphocytes immediately after surgery [48]

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All in all, the authors proposed that long periods of elevated reactive oxygen species damage the mitochondria, producing a long-term problem of energy depletion

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