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Frei, M

The survival of T-ALL cells after DOX treatment was correlated with the expression of Mitofusin-2 (MFN-2)A GTPase protein that mediates mitochondrial fusionWith an increase in the expression of CTE complexes as well as a proportional increase in oxygen consumption rate (OCR), and increased sensitivity to DOX was evident upon inactivating MFN-2 by CRISPR ( The anti-apoptotic protein Bcl-2 homologous antagonist/killer (BCL-2), overexpressed in some subtypes of B-cell lymphomas, regulates mitochondrial function and mediates therapeutic resistance by inhibiting the oligomerization of the pro-apoptotic Bcl-2 homologous antagonist/killer (Bak) and Bcl-2-associated X protein (Bax) proteins and by facilitating the import of GSH and complex IV subunits to mitochondria (Mattes et al., 2019)

As the signal is amplified, so is the amount of phosphorylation leading to an accentuation of signal amplification

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For other parenteral routes such as subcutaneous and intramuscular injections, the bioavailability may still be close to 100 % for most therapeutic drugs (F = 0.751), since little or no significant metabolism of the drug occurs in the skin or muscle and these small molecules (MW 0 min) compared to the intravenous route