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The use of human MNCs was approved by the Ethics Committee of Kiel University under informed consent from healthy donors

The selection and use of peptides are highly individual, and what works for one person may not work for another

Glucose metabolism has been shown to modulate the effector function of T H 17 cells by controlling a metabolic-transcriptional network, with Acly serving as a key rate-limiting mediator generating acetyl-CoA and dictating T H 17 cell pathogenicity 21 , while restraining the development of FOXP3 + T regs 74

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3.2 Mitochondrial metabolites as immune signaling molecules In HIV infection, mitochondrial dysfunction alters the levels of key metabolic intermediates, converting them from homeostatic signaling molecules into drivers of pathology