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In addition to these interactions, Gln3 of oxyntomodulin formed transient hydrogen bonds and hydrophobic contacts with T391 7.46 and E387 7.42 , as well as van der Waals interactions with L388 7.43 , while Glu9 in GLP-1 only formed hydrophobic interactions with these residues

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Replacement of the GLP-2 middle region (residues 719) that mainly interacts with TM1, TM7 and ECL1 by the corresponding part of GLP-1, almost abolished GLP-2R-mediated cAMP accumulation but fully rescued that induced by GLP-1R (Fig

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CD4 + T cells contribute to the exacerbation of cerebral inflammation and the induction of neuronal death (74, 75)