However, a dysregulated increase in the level of these free radicals, as seen in mitochondrial diseases, can result in oxidative damage by overwhelming the cellular antioxidant defence mechanism, of which glutathione is the principal component [4]
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W badaniach laboratoryjnych wykazano, e GHK-Cu moe chroni komrki nerwowe przed toksycznym dziaaniem nadmiaru glutaminianu, co jest wane w kontekcie chorb neurodegeneracyjnych, takich jak choroba Alzheimera czy Parkinsona