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In adaptive immunity, PXR functions predominantly as a negative regulator by suppressing T lymphocyte activation and proliferation, limiting interferon- (IFN-) production, and restraining B-1 cell development (82, 83)

Likewise, maintaining modulation and reestablishing normal system functioning may occur as the counteraction of NO-synthase (NOS)-substrate over-activity (L-arginine-hypotension and anti-thrombotic effect counteracted) [98,100]

O'Connor 2015

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