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FAT OXIDATION AND LIPID METABOLISM OPTIMIZATION With enhanced mitochondrial capacity and SIRT1-mediated metabolic optimization, cells preferentially oxidize fat for energy

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Action: This long half-life allows it to drive muscle hyperplasia (new cell creation) and hypertrophy (cell size increase) over a sustained period by binding to the IGF-1 Receptor (IGF-1R) with high affinity

Esta heterogeneidad refleja la posicin intermedia de este pptido, que no es ni un medicamento aprobado ni una sustancia estrictamente controlada en la mayora de los pases