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Preclinical models demonstrate that region-specific elevations of central KYNA highlight a functional trade-off: while KYNA is often considered neuroprotective due to its antagonism of glutamatergic receptors, its broad receptor profile also allows it to inhibit 7nAChRs, which are essential for prefrontal cortical processing and mood regulation

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10.3390/ijms25115640 [DOI] [PMC free article] [PubMed] [Google Scholar] Qi L., Groeger M., Sharma A., Goswami I., Chen E., Zhong F., et al

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Inflammation Modulation Studied for their potential to regulate inflammatory pathways in experimental models