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Possible mechanisms to account for the protection exerted by vaginal lactobacilli include inactivation of pathogens by different metabolic Lactobacillus products (lactic acid, H2O2, bacteriocins), competition for epithelial cell attachment sites, preservation of mucin gel coating the vaginal/cervical epithelium through inhibition of glycosidase-producing anaerobes, and maintenance of appropriate innate immune response (14-16)

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