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DPP IV inhibitor suppresses STZ-induced islets injury dependent on activation of the IGFR/Akt/mTOR signaling pathways by GLP-1 in monkeys

Proinflammatory switch from Galphas to Galphai signaling by Glucagon-like peptide-1 receptor in murine splenic monocyte following burn injury

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GLP-1 receptor agonists work by mimicking the action of naturally occurring GLP-1, a hormone that stimulates insulin secretion in response to food intake, suppresses glucagon release, slows gastric emptying, and promotes satiety

[ There is still a controversy whether high UA is a compensatory mechanism to overcome increased oxidative stress, which is an independent cause of cardiovascular disease [ and is a component of the inflammatory process