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These systemic benefits reflect BPC-157s broad regenerative properties

Our results provide a possible mechanism in which upregulation of phosphorylated p53 and p16 maintain the cell cycle arrest of keloid fibroblasts, and FOXO4-DRI selectively induces apoptosis of keloid senescent fibroblasts by prompting p53-pS15 nuclear exclusion

Similarly, neural therapy could offer additional benefits depending on individual circumstances

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Interestingly, this effect appears to be context-dependent, that is, robust in vivo, but not consistently observed in isolated in vitro models